The Effect of Mucosidosis (Cystic Fibrosis) and Bacterial Factors on CFTR (CF Transmembrane Conductance Regulator) Channel Efficiency: OAJBS Publishers

The Effect of Mucosidosis (Cystic Fibrosis) and Bacterial Factors on CFTR (CF Transmembrane Conductance Regulator) Channel Efficiency by Ali Ahmadi* in Open Access Journal of Biomedical Science (OAJBS)

Mucocidosis (cystic fibrosis) is the most common autosomal recessive inherited multisystem disease with a very deadly and dangerous outcome due to mutations and sequential mutations in the gene regulating the transmission of cystic fibrosis membrane (CFTR) leading to the chloride channel. And the CFTR protein becomes unhealthy and destructive [1,2]. As a result of the collection and collection of mucus that is not sufficiently hydrated and hydrophilic, it affects various basic organs, mainly lungs, and airways, pancreas, liver, bile ducts and intestines. This leads to inflammation and infection, fibrosis, and gradual tissue destruction [3-5]. Respiratory failure and subsequent problems have been cited as one of the leading causes of death. However, more than 30 years of molecular description A major defect in CFTR causes CF, a remarkable success in the long-term prognosis of people with CF. This breakthrough is very active in the spirit of collaboration and networking of the CF research community, and internationally, with the creation of a multidisciplinary clinical CF team that implements evidence in various aspects of standard care with the CF patient has been obtained. Rath et al. [5]. In their study found that several bacteria and fungi that cause organisms are also known as urinary tract infections (UTIs) that may occur in more than 50% of the population and are more common in women [5-8].
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